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Knockout-Transgenic Mouse Model of Sickle Cell Disease
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Homocysteine Increases the Expression of Vascular Endothelial Growth Factor by a Mechanism Involving Endoplasmic Reticulum Stress and Transcription Factor ATF4
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Oxygen Radical Inhibition of Nitric Oxide-Dependent Vascular Function in Sickle Cell Disease
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Induction of KLF4 in basal keratinocytes blocks the proliferation-differentiation switch and initiates squamous epithelial dysplasia
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The bZIP transcription factor LCR-F1 is essential for mesoderm formation in mouse development
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Cloning and functional characterization of LCR-F1: a bZIP transcription factor that activates erythroid-specific, human globin gene expression
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Published in Nucleic acids research (25.06.1994)
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A single erythroid-specific DNase I super-hypersensitive site activates high levels of human β-globin gene expression in transgenic mice
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Published in Genes & development (01.03.1989)
Published in Genes & development (01.03.1989)
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Human γ- to β-globin gene switching in transgenic mice
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Published in Genes & development (01.03.1990)
Published in Genes & development (01.03.1990)
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Erythroid‐specific expression of human beta‐globin genes in transgenic mice
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Published in The EMBO journal (01.07.1985)
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Published in Circulation (New York, N.Y.) (04.06.2019)
Published in Circulation (New York, N.Y.) (04.06.2019)
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