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Immune complexes from SLE sera induce IL10 production from normal peripheral blood mononuclear cells by an FcγRII dependent mechanism: implications for a possible vicious cycle maintaining B cell hyperactivity in SLE

by Rönnelid, J, Tejde, A, Mathsson, L, Nilsson-Ekdahl, K, Nilsson, B
Published in Annals of the rheumatic diseases (01.01.2003)

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Immune complex‐stimulated production of interleukin‐12 in peripheral blood mononuclear cells is regulated by the complement system

by TEJDE, A., MATHSSON, L., EKDAHL, K. N., NILSSON, B., RÖNNELID, J.
Published in Clinical and experimental immunology (01.09.2004)

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Cryoglobulin‐induced cytokine production via FcγRIIa: inverse effects of complement blockade on the production of TNF‐α and IL‐10. Implications for the growth of malignant B‐cell clones

by Mathsson, Linda, Tejde, Andreas, Carlson, Kristina, Höglund, Martin, Nilsson, Bo, Nilsson‐Ekdahl, Kristina, Rönnelid, Johan
Published in British journal of haematology (01.06.2005)

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Immune complexes from SLE sera induce IL10 production from normal peripheral blood mononuclear cells by an Fc[GAMMA]RII dependent mechanism: implications for a possible vicious cycle maintaining B cell hyperactivity in SLE

by Rönnelid, J, Tejde, A, Mathsson, L, Nilsson-Ekdahl, K, Nilsson, B
Published in Annals of the rheumatic diseases (01.01.2003)

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Immune complexes from SLE sera induce IL10 production from normal peripheral blood mononuclear cells by an FcgammaRII dependent mechanism: implications for a possible vicious cycle maintaining B cell hyperactivity in SLE

by Rönnelid, J, Tejde, A, Mathsson, L, Nilsson-Ekdahl, K, Nilsson, B
Published in Annals of the rheumatic diseases (01.01.2003)

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Immune complexes from SLE sera induce IL-10 production by a Fc-RII-dependent mechanism: A possible vicious circle maintaining B cell hyperactivity in SLE

by Rönnelid, J, Tejde, A, Mathsson, L, Nilsson Ekdahl, Kristina, Nilsson, Bo
Published in Annals of the rheumatic diseases (2003)

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Immune complex stimulation of peripheral blood mononuclear cells result in enhancement of suppression of IL-12 production dependent on soluble serum factors

by Tejde, A, Nilsson Ekdahl, K, Nilsson, B, Rönnelid, J
Published in Arthritis research (26.01.2001)

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Immune complexes from SLE sera induce IL10 production from normal peripheral blood mononuclear cells by an FcγRII dependent mechanism: implications for a possible vicious cycle maintaining B cell hyperactivity in SLE

Immune complex‐stimulated production of interleukin‐12 in peripheral blood mononuclear cells is regulated by the complement system

Cryoglobulin‐induced cytokine production via FcγRIIa: inverse effects of complement blockade on the production of TNF‐α and IL‐10. Implications for the growth of malignant B‐cell clones

Immune complexes from SLE sera induce IL10 production from normal peripheral blood mononuclear cells by an Fc[GAMMA]RII dependent mechanism: implications for a possible vicious cycle maintaining B cell hyperactivity in SLE

Immune complexes from SLE sera induce IL10 production from normal peripheral blood mononuclear cells by an FcgammaRII dependent mechanism: implications for a possible vicious cycle maintaining B cell hyperactivity in SLE

Immune complexes from SLE sera induce IL-10 production by a Fc-RII-dependent mechanism: A possible vicious circle maintaining B cell hyperactivity in SLE

Immune complex stimulation of peripheral blood mononuclear cells result in enhancement of suppression of IL-12 production dependent on soluble serum factors

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