Search Results - "McIntosh, Rachel A." :: K.UTB vyhledávací portál

Exogenous zinc protects cardiac cells from reperfusion injury by targeting mitochondrial permeability transition pore through inactivation of glycogen synthase kinase-3β

by Chanoit, Guillaume, Lee, SungRyul, Xi, Jinkun, Zhu, Min, McIntosh, Rachel A., Mueller, Robert A., Norfleet, Edward A., Xu, Zhelong
Published in American journal of physiology. Heart and circulatory physiology (01.09.2008)

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Exogenous zinc protects cardiac cells from reperfusion injury by targeting mitochondrial permeability transition pore through inactivation of glycogen synthase kinase-3beta

by Chanoit, Guillaume, Lee, SungRyul, Xi, Jinkun, Zhu, Min, McIntosh, Rachel A, Mueller, Robert A, Norfleet, Edward A, Xu, Zhelong
Published in American journal of physiology. Heart and circulatory physiology (01.09.2008)

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Exogenous zinc protects cardiac cells from reperfusion injury by targeting mitochondrial permeability transition pore through inactivation of glycogen synthase kinase-3{beta}

by Chanoit, Guillaume, Lee, SungRyul, Xi, Jinkun, Zhu, Min, McIntosh, Rachel A, Mueller, Robert A, Norfleet, Edward A, Xu, Zhelong
Published in American journal of physiology. Heart and circulatory physiology (01.09.2008)

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Exogenous zinc protects cardiac cells from reperfusion injury by targeting mitochondrial permeability transition pore through inactivation of glycogen synthase kinase-3β

by CHANOIT, Guillaume, LEE, Sungryul, JINKUN XI, MIN ZHU, MCINTOSH, Rachel A, MUELLER, Robert A, NORFLEET, Edward A, ZHELONG XU
Published in American journal of physiology. Heart and circulatory physiology (2008)

Get full text

Journal Article

Exogenous zinc protects cardiac cells from reperfusion injury by targeting mitochondrial permeability transition pore through inactivation of glycogen synthase kinase-3β

Exogenous zinc protects cardiac cells from reperfusion injury by targeting mitochondrial permeability transition pore through inactivation of glycogen synthase kinase-3beta

Exogenous zinc protects cardiac cells from reperfusion injury by targeting mitochondrial permeability transition pore through inactivation of glycogen synthase kinase-3{beta}

Exogenous zinc protects cardiac cells from reperfusion injury by targeting mitochondrial permeability transition pore through inactivation of glycogen synthase kinase-3β

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