Search Results - "KLIPPEL, ANKE" :: K.UTB vyhledávací portál | TBU Library Retrieval Portal

CC-122, a pleiotropic pathway modifier, mimics an interferon response and has antitumor activity in DLBCL

CC-122, a pleiotropic pathway modifier, mimics an interferon response and has antitumor activity in DLBCL

by Hagner, Patrick R., Man, Hon-Wah, Fontanillo, Celia, Wang, Maria, Couto, Suzana, Breider, Mike, Bjorklund, Chad, Havens, Courtney G., Lu, Gang, Rychak, Emily, Raymon, Heather, Narla, Rama Krishna, Barnes, Leo, Khambatta, Gody, Chiu, Hsiling, Kosek, Jolanta, Kang, Jian, Amantangelo, Michael D., Waldman, Michelle, Lopez-Girona, Antonia, Cai, Ti, Pourdehnad, Michael, Trotter, Matthew, Daniel, Thomas O., Schafer, Peter H., Klippel, Anke, Thakurta, Anjan, Chopra, Rajesh, Gandhi, Anita K.
Published in Blood (06.08.2015)

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Regulation of epidermal homeostasis and repair by phosphoinositide 3-kinase

Regulation of epidermal homeostasis and repair by phosphoinositide 3-kinase

by Pankow, Sandra, Bamberger, Casimir, Klippel, Anke, Werner, Sabine
Published in Journal of cell science (01.10.2006)

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Peroxiredoxin 6 is required for blood vessel integrity in wounded skin

Peroxiredoxin 6 is required for blood vessel integrity in wounded skin

by Kümin, Angelika, Schäfer, Matthias, Epp, Nikolas, Bugnon, Philippe, Born-Berclaz, Christiane, Oxenius, Annette, Klippel, Anke, Bloch, Wilhelm, Werner, Sabine
Published in The Journal of cell biology (19.11.2007)

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REDD1 integrates hypoxia-mediated survival signaling downstream of phosphatidylinositol 3-kinase

REDD1 integrates hypoxia-mediated survival signaling downstream of phosphatidylinositol 3-kinase

by SCHWARZER, Rolf, TONDERA, Daniel, ARNOLD, Wolfgang, GIESE, Klaus, KLIPPEL, Anke, KAUFMANN, Jörg
Published in Oncogene (10.02.2005)

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Differential regulation of TGF-β signaling through Smad2, Smad3 and Smad4

Differential regulation of TGF-β signaling through Smad2, Smad3 and Smad4

by KRETSCHMER, Anny, MOEPERT, Kristin, DAMES, Sibylle, STERNBERGER, Maria, KAUFMANN, Joerg, KLIPPEL, Anke
Published in Oncogene (02.10.2003)

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Knockdown of MTP18, a Novel Phosphatidylinositol 3-Kinase-dependent Protein, Affects Mitochondrial Morphology and Induces Apoptosis

Knockdown of MTP18, a Novel Phosphatidylinositol 3-Kinase-dependent Protein, Affects Mitochondrial Morphology and Induces Apoptosis

by Tondera, Daniel, Santel, Ansgar, Schwarzer, Rolf, Dames, Sybille, Giese, Klaus, Klippel, Anke, Kaufmann, Jörg
Published in The Journal of biological chemistry (23.07.2004)

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Enzyme kinetics and distinct modulation of the protein kinase N family of kinases by lipid activators and small molecule inhibitors

Enzyme kinetics and distinct modulation of the protein kinase N family of kinases by lipid activators and small molecule inhibitors

by Falk, Matthew D, Liu, Wei, Bolaños, Ben, Unsal-Kacmaz, Keziban, Klippel, Anke, Grant, Stephan, Brooun, Alexei, Timofeevski, Sergei
Published in Bioscience reports (01.04.2014)

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Caspase-3-Generated Fragment of Gelsolin: Effector of Morphological Change in Apoptosis

Caspase-3-Generated Fragment of Gelsolin: Effector of Morphological Change in Apoptosis

by Kothakota, Srinivas, Azuma, Toshifumi, Reinhard, Christoph, Klippel, Anke, Tang, Jay, Chu, Keting, McGarry, Thomas J., Kirschner, Marc W., Koths, Kirston, Kwiatkowski, David J., Williams, Lewis T.
Published in Science (American Association for the Advancement of Science) (10.10.1997)

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A specific product of phosphatidylinositol 3-kinase directly activates the protein kinase Akt through its pleckstrin homology domain

A specific product of phosphatidylinositol 3-kinase directly activates the protein kinase Akt through its pleckstrin homology domain

by Klippel, Anke, Kavanaugh, W. Michael, Pot, David, Williams, Lewis T.
Published in Molecular and Cellular Biology (01.01.1997)

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Smad3 Is a Key Nonredundant Mediator of Transforming Growth Factor β Signaling in Nme Mouse Mammary Epithelial Cells

Smad3 Is a Key Nonredundant Mediator of Transforming Growth Factor β Signaling in Nme Mouse Mammary Epithelial Cells

by Dzwonek, Joanna, Preobrazhenska, Olena, Cazzola, Silvia, Conidi, Andrea, Schellens, Ann, van Dinther, Maarten, Stubbs, Andrew, Klippel, Anke, Huylebroeck, Danny, ten Dijke, Peter, Verschueren, Kristin
Published in Molecular cancer research (01.08.2009)

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Membrane localization of phosphatidylinositol 3-kinase is sufficient to activate multiple signal-transducing kinase pathways

Membrane localization of phosphatidylinositol 3-kinase is sufficient to activate multiple signal-transducing kinase pathways

by Klippel, Anke, Reinhard, Christoph, Kavanaugh, W. Michael, Apell, Gerald, Escobedo, Maria-Amelia, Williams, Lewis T.
Published in Molecular and Cellular Biology (01.08.1996)

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Activation of Phosphatidylinositol 3-Kinase Is Sufficient for Cell Cycle Entry and Promotes Cellular Changes Characteristic of Oncogenic Transformation

Activation of Phosphatidylinositol 3-Kinase Is Sufficient for Cell Cycle Entry and Promotes Cellular Changes Characteristic of Oncogenic Transformation

by Klippel, Anke, Escobedo, Maria-Amelia, Wachowicz, Matthew S., Apell, Gerald, Brown, Timothy W., Giedlin, Martin A., Kavanaugh, W. Michael, Williams, Lewis T.
Published in Molecular and Cellular Biology (01.10.1998)

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Activated Phosphatidylinositol 3-Kinase and Akt Kinase Promote Survival of Superior Cervical Neurons

Activated Phosphatidylinositol 3-Kinase and Akt Kinase Promote Survival of Superior Cervical Neurons

by Philpott, Karen L., McCarthy, Mary Jane, Klippel, Anke, Rubin, Lee L.
Published in The Journal of cell biology (03.11.1997)

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Ras-Dependent Induction of Cellular Responses by Constitutively Active Phosphatidylinositol-3 Kinase

Ras-Dependent Induction of Cellular Responses by Constitutively Active Phosphatidylinositol-3 Kinase

by Hu, Qianjin, Klippel, Anke, Muslin, Anthony J., Fantl, Wendy J., Williams, Lewis T.
Published in Science (American Association for the Advancement of Science) (07.04.1995)

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Activated Phosphatidylinositol 3-Kinase Is Sufficient to Mediate Actin Rearrangement and GLUT4 Translocation in 3T3-L1 Adipocytes

Activated Phosphatidylinositol 3-Kinase Is Sufficient to Mediate Actin Rearrangement and GLUT4 Translocation in 3T3-L1 Adipocytes

by Martin, S S, Haruta, T, Morris, A J, Klippel, A, Williams, L T, Olefsky, J M
Published in The Journal of biological chemistry (26.07.1996)

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Protein Kinase B/Akt Mediates Effects of Insulin on Hepatic Insulin-like Growth Factor-binding Protein-1 Gene Expression through a Conserved Insulin Response Sequence

Protein Kinase B/Akt Mediates Effects of Insulin on Hepatic Insulin-like Growth Factor-binding Protein-1 Gene Expression through a Conserved Insulin Response Sequence

by Cichy, S B, Uddin, S, Danilkovich, A, Guo, S, Klippel, A, Unterman, T G
Published in The Journal of biological chemistry (13.03.1998)

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Overexpression of a Constitutively Active Form of Phosphatidylinositol 3-Kinase Is Sufficient to Promote Glut 4 Translocation in Adipocytes

Overexpression of a Constitutively Active Form of Phosphatidylinositol 3-Kinase Is Sufficient to Promote Glut 4 Translocation in Adipocytes

by Tanti, Jean-François, Grémeaux, Thierry, Grillo, Sophie, Calleja, Véronique, Klippel, Anke, Williams, Lewis T., Van Obberghen, Emmanuel, Le Marchand-Brustel, Yannick
Published in The Journal of biological chemistry (11.10.1996)

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MCT4 is an important determinant for the growth of highly glycolytic and aggressive malignancies

MCT4 is an important determinant for the growth of highly glycolytic and aggressive malignancies

by Unsal-Kacmaz, Kezi, Ragunathan, Shoba, Shan, Jennifer, Guo, Bing, Wang, Xiaoli, Tkach, Diane, Lucas, Judy, Klippel, Anke
Published in BMC proceedings (01.06.2012)

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Cross-talk between Phosphatidylinositol 3-Kinase and Sphingomyelinase Pathways as a Mechanism for Cell Survival/Death Decisions

Cross-talk between Phosphatidylinositol 3-Kinase and Sphingomyelinase Pathways as a Mechanism for Cell Survival/Death Decisions

by Burow, Matthew E., Weldon, Christopher B., Collins-Burow, Bridgette M., Ramsey, Nijm, McKee, Amy, Klippel, Anke, McLachlan, John A., Clejan, Sanda, Beckman, Barbara S.
Published in The Journal of biological chemistry (31.03.2000)

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Immunomodulatory agents lenalidomide and pomalidomide co-stimulate T cells by inducing degradation of T cell repressors Ikaros and Aiolos via modulation of the E3 ubiquitin ligase complex CRL4(CRBN.)

Immunomodulatory agents lenalidomide and pomalidomide co-stimulate T cells by inducing degradation of T cell repressors Ikaros and Aiolos via modulation of the E3 ubiquitin ligase complex CRL4(CRBN.)

by Gandhi, Anita K, Kang, Jian, Havens, Courtney G, Conklin, Thomas, Ning, Yuhong, Wu, Lei, Ito, Takumi, Ando, Hideki, Waldman, Michelle F, Thakurta, Anjan, Klippel, Anke, Handa, Hiroshi, Daniel, Thomas O, Schafer, Peter H, Chopra, Rajesh
Published in British journal of haematology (01.03.2014)

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