The tarantula toxins ProTx-II and huwentoxin-IV differentially interact with human Nav1.7 voltage sensors to inhibit channel activation and inactivation
Xiao, Yucheng, Blumenthal, Kenneth, Jackson, 2nd, James O, Liang, Songping, Cummins, Theodore R
Published in Molecular pharmacology (01.12.2010)
Published in Molecular pharmacology (01.12.2010)
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Human voltage-gated sodium channel mutations that cause inherited neuronal and muscle channelopathies increase resurgent sodium currents
Jarecki, Brian W, Piekarz, Andrew D, Jackson, 2nd, James O, Cummins, Theodore R
Published in The Journal of clinical investigation (01.01.2010)
Published in The Journal of clinical investigation (01.01.2010)
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Gas discharge plasmas are effective in inactivating Bacillus and Clostridium spores
Tseng, Shawn, Abramzon, Nina, Jackson, James O., Lin, Wei-Jen
Published in Applied microbiology and biotechnology (01.03.2012)
Published in Applied microbiology and biotechnology (01.03.2012)
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A Nav1.7 channel mutation associated with hereditary erythromelalgia contributes to neuronal hyperexcitability and displays reduced lidocaine sensitivity
Sheets, Patrick L., Jackson, James O., Waxman, Stephen G., Dib‐Hajj, Sulayman D., Cummins, Theodore R.
Published in The Journal of physiology (15.06.2007)
Published in The Journal of physiology (15.06.2007)
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The Tarantula Toxins ProTx-II and Huwentoxin-IV Differentially Interact with Human Na v 1.7 Voltage Sensors to Inhibit Channel Activation and Inactivation
Xiao, Yucheng, Blumenthal, Kenneth, Jackson, James O., Liang, Songping, Cummins, Theodore R.
Published in Molecular pharmacology (01.12.2010)
Published in Molecular pharmacology (01.12.2010)
Get full text
Journal Article
The Tarantula Toxins ProTx-II and Huwentoxin-IV Differentially Interact with Human Nav1.7 Voltage Sensors to Inhibit Channel Activation and InactivationS
Xiao, Yucheng, Blumenthal, Kenneth, Jackson, James O., Liang, Songping, Cummins, Theodore R.
Published in Molecular pharmacology (01.12.2010)
Published in Molecular pharmacology (01.12.2010)
Get full text
Journal Article
A Na v 1.7 channel mutation associated with hereditary erythromelalgia contributes to neuronal hyperexcitability and displays reduced lidocaine sensitivity
Sheets, Patrick L., Jackson, James O., Waxman, Stephen G., Dib‐Hajj, Sulayman D., Cummins, Theodore R.
Published in The Journal of physiology (15.06.2007)
Published in The Journal of physiology (15.06.2007)
Get full text
Journal Article
A Na sub(v)1.7 channel mutation associated with hereditary erythromelalgia contributes to neuronal hyperexcitability and displays reduced lidocaine sensitivity
Sheets, Patrick L, Jackson, James O, Waxman, Stephen G, Dib-Hajj, Sulayman D, Cummins, Theodore R
Published in The Journal of physiology (15.06.2007)
Published in The Journal of physiology (15.06.2007)
Get full text
Journal Article