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High sucrose consumption induces memory impairment in rats associated with electrophysiological modifications but not with metabolic changes in the hippocampus
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Adenosine A2A receptor facilitation of hippocampal synaptic transmission is dependent on tonic A1 receptor inhibition
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Adenosine A2A receptors and basal ganglia physiology
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Optogenetic activation of intracellular adenosine [A.sub.2A] receptor signaling in the hippocampus is sufficient to trigger CREB phosphorylation and impair memory
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